Science Signaling Podcast
Summary: Periodic audiocasts from Science Signaling, the leading journal of regulatory biology and cell signaling in physiology and disease
- Visit Website
- RSS
- Artist: Science Signaling
- Copyright: © 2017 American Association for the Advancement of Science
Podcasts:
Javier González-Maeso explains how ligand binding to one member of a heteromeric GPCR complex can activate its binding partner.
Science Signaling editor Nancy Gough and Chief Scientific Editor Michael Yaffe discuss the current state of signaling research and the journal's place in this dynamic landscape.
Carl White explains how the gaseous signaling molecule hydrogen sulfide suppresses calcium signaling in adipose tissue macrophages to limit inflammation.
Theo Rein explains how the antidepressant paroxetine affects epigenetic regulation of Bdnf.
Susanne Muehlich describes how actin dynamics control activity of the transcriptional coactivator MKL1.
Moritz Bünemann and Peter Kolb explain how the mode of ligand binding to a G protein–coupled receptor determines whether changes in membrane potential enhance or attenuate signaling.
Fengyuan Tang has demonstrated that the kinases NDR1 and NDR2 mediate Hippo signaling during thymocyte migration.
Valérie Dardalhon and Naomi Taylor explain how availability of the nutrient glutamine influences the differentiation of T cells in the immune system.
Lois Smith and Ye Sun explain why SOCS3 activity in neural and glial cells of the retina prevents pathological retinal neovascularization.
Roy Silverstein explains how the Na+/K+-ATPase helps turn macrophages into foam cells at atherosclerotic plaques.
Markus Gerhard explains how the E3 ubiquitin ligase RNF43 inhibits a transcription factor that mediates Wnt-β-catenin signaling without ubiquitylating it.
Todd Rosen and Bingbing Wang report on an epigenetic mechanism by which glucocorticoids trigger placental expression of a stress hormone associated with labor.
Sylvain Galvani and Timothy Hla explain why the lipid S1P protects blood vessels when it is bound to HDL.
Pooja Desai explains that distinct calcium channel pore subunits produced from the same transcript mediate different types of calcium currents.
Cristina Murga and Rocio Vila-Bedmar explain why removing the kinase GRK2 prevents some of the metabolic consequences of a high-fat diet.